Bell's palsy affects pregnant women three times more often than nonpregnant women, with majority of cases occurring in the third trimester (71%) and early postpartum period (21%). Bell's palsy during pregnancy has been associated with preeclampsia, with 22% of these women developing preeclampsia. A 26-year-old G3P1L1A1 presented at 33 weeks period of gestation with chief complaint of progressive bilateral pedal edema since a week, blood pressure (BP) 150/100 mm Hg, dipstick urine 3+. Labetalol 100 mg 12 hourly was commenced for control of BP. Laboratory screening of HELLP (hemolysis, elevated liver enzymes, and low platelet count) syndrome was normal. Six days later she had an episode of generalized tonic–clonic convulsion, magnesium sulfate was given and emergency laboratory investigations revealed partial HELLP syndrome [platelet 80,000, serum glutamic oxaloacetic transaminase (SGOT)/serum glutamic pyruvic transaminase (SGPT) 281/222 U/L] with normal coagulation profile. Lower segment cesarean section was done under general anesthesia with delivery of a 2 kg baby with Apgar score of 7/10 and 9/10 at 1 and 5 minutes respectively. Postoperative day 3, all laboratory reports including magnetic resonance imaging (MRI) brain were normal. Blood pressure was stabilized and she was discharged on third postnatal day, with some right-sided residual facial weakness. All obstetricians must be aware of association of Bell's palsy with hypertensive disorders of pregnancy and aggressive screen for the same as soon as diagnosed, in order to prevent complications like eclampsia and HELLP. Since Bell's palsy is not associated with adverse perinatal outcomes, it should not prompt obstetricians for unnecessary preterm inductions.